Cognitive Effects
Beginning in the thirties is point in the aging process when it is typical to experience cognitive declines. However it is possible to maintain normal cognitive function into old age. Many elderly have memories as sharp or even sharper than their younger counterparts. Semantic or general knowledge memory including vocabulary and definitions remain steady or even increase.
Emotional Effects
Aging typically produces and improvement in emotional intelligence. This is because with age we typically are better able to manage and regulate our emotions. The exception is usually related to health and wellness deficiencies, disease states, or prolonged stress.
Successful Ageing
The Golden Years have been termed "successful ageing" as early as the 1950s and was popularized in the late 80s. The terminology has sparked a debate as to what is “normal ageing" which would have a high risk of illness and "successful ageing" with low risk of disability and high cognitive and physical functioning.
Successful ageing would consist of three components:
1. Low probability of disease or disability
2. High mental and physical function capacity
3. Active engagement with life Alternatively, terms such as "healthy ageing," "optimal ageing" have been proposed.
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Showing posts with label Aging. Show all posts
Showing posts with label Aging. Show all posts
Tuesday, February 12, 2008
The Ultimate Anti-aging Treatment
To facilitate anti aging requires understanding aging. With this knowledge making the changes in life style and diet that can enhance the anti aging process.
Aging
In health care, Ageing or aging in the human body refers to the deterioration of the body the longer we live.
There are social, cognitive, cultural, and economic effects that result from and are effects of Aging. These can affect the way we live our lives, the quality of life we have and the way society views us. One of the ways we care for aging is anti aging treatments.
The Aging population is an important issue in many nations, especially those North of the equator. Thus, age has in influence on the functioning of the society we live in.
In science and biology, Aging is divided into cellular and organism Aging.
The Aging of an organism is is often noted by the declining ability to respond to stress, increased imbalanced homeostasis and increased risk of disease. Some researchers are treating aging as disease. As such, it is considered treatable.
Diet has been shown to play a large role in the anti aging promotion. Dietary intake is considered in a number of ways including nutrient intake, quantity of intake and toxic exposure.
Click here for more about Glutathione and its benefits to our body.
Aging
In health care, Ageing or aging in the human body refers to the deterioration of the body the longer we live.
There are social, cognitive, cultural, and economic effects that result from and are effects of Aging. These can affect the way we live our lives, the quality of life we have and the way society views us. One of the ways we care for aging is anti aging treatments.
The Aging population is an important issue in many nations, especially those North of the equator. Thus, age has in influence on the functioning of the society we live in.
In science and biology, Aging is divided into cellular and organism Aging.
The Aging of an organism is is often noted by the declining ability to respond to stress, increased imbalanced homeostasis and increased risk of disease. Some researchers are treating aging as disease. As such, it is considered treatable.
Diet has been shown to play a large role in the anti aging promotion. Dietary intake is considered in a number of ways including nutrient intake, quantity of intake and toxic exposure.
Click here for more about Glutathione and its benefits to our body.
Monday, February 11, 2008
Antioxidants, and the Immune System
The proliferation of T and B cells, natural killer cells, and lymphokine activated killer cells that are required to mount an effective defense against pathogens and tumor cells appear to be inhibited markedly with age and upon exposure to oxidants. These effects can, in part, be counteracted in elderly individuals by dietary antioxidant supplementation. While the endogenous sources of oxidants that lead to the suppression of lymphocyte dependent immunity are not known, in vitro studies have demonstrated that polymorphonuclear leucocytes and macrophages, both can inhibit proliferation of various lymphocyte subpopulations through the production of reactive oxygen intermediates and the prostaglandin metabolite PGE2 as well as from nitric oxide.
This suggests that conditions that involve infiltration of polymorphonuclear leucocytes and macrophages (i.e., chronic inflammatory diseases), could result in compromised lymphocyte function. The suppressive effects of macrophages on mitogen induced lymphocyte proliferation can be reversed partially by thiol reagents, catalase or indomethacin, or by NG-monomethyl-L-arginine, a competitive inhibitor of nitric oxide synthesis.
The age associated decrease in cell mediated immunity may be due to a decreased level of certain small molecule antioxidant and antioxidant enzymes that accompany the aging process.
Calorie restriction, a dietary regimen that increases maximal lifespan in rodents also enhances T lymphocyte responsiveness possibly by slowing the rate of thymus involution and by boosting the level of cellular antioxidant defenses.
This suggests that conditions that involve infiltration of polymorphonuclear leucocytes and macrophages (i.e., chronic inflammatory diseases), could result in compromised lymphocyte function. The suppressive effects of macrophages on mitogen induced lymphocyte proliferation can be reversed partially by thiol reagents, catalase or indomethacin, or by NG-monomethyl-L-arginine, a competitive inhibitor of nitric oxide synthesis.
The age associated decrease in cell mediated immunity may be due to a decreased level of certain small molecule antioxidant and antioxidant enzymes that accompany the aging process.
Calorie restriction, a dietary regimen that increases maximal lifespan in rodents also enhances T lymphocyte responsiveness possibly by slowing the rate of thymus involution and by boosting the level of cellular antioxidant defenses.
Aging is slowed by calorie or protein restriction
In rodents a calorie restricted diet, significantly increases lifespan, decreases reproduction, and markedly decreases cancer rates.
It has been suggested that Darwinian fitness in animals is increased by the delay of reproductive function during periods of low food availability and that the saved resources are invested in maintenance of the body until food resources are available for successful reproduction.
Protein restriction appears to have the same effects on rodents as calorie restriction, though it is less well-studied. An understanding of mechanisms for this marked effect on aging and cancer is becoming clearer and may in good part be due to reduced oxidative damage.
The suggestion that maintenance functions are enhanced in calorie-restricted rats thus resulting in less oxidative damage is supported by the findings of more efficient DNA repair, better coupled mitochondrial respiration and a delay in the age-dependent decline of antioxidant defenses.
The higher level of antioxidant defenses could also account for the enhanced immune response in restricted animals. We have recently shown that either calorie or protein restriction decreases the rate of accumulation of oxidized protein that accompanies aging in rats and preliminary results suggest a decrease in preneoplastic foci and oxidative lesions in DNA as well.
Thus, the overall effect of these enhanced maintenance activities appears to be a reduction in oxidative damage to DNA and protein, a decrease in DNA and protein lesions, and a decrease in somatic mutations. Markedly lower mitotic rates are observed in a variety of tissues in calorie restricted compared to ad libitum fed rodent, which may also contribute to the decrease in tumor incidence.
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*Dietary restriction activates the pituitary adrenocorticotropic axis resulting in a decrease in the release of reproductive and mitogenic hormones. Decreases in mitogenic hormones such as insulin, TSH, growth hormone, estrogen, and prolactin decrease the likelihood of hormone-induced cancers, as has been shown in various animal studies. This is consistent with suppression of mitogenic hormones and decreased proto-oncogene expression. The lowered incidence of mammary tumors observed in calorie-restricted rats has been attributed to reduced circulating levels of the mammotropic hormones estrogen and prolactin.
Click here for more about Glutathione and its benefits to our body.
It has been suggested that Darwinian fitness in animals is increased by the delay of reproductive function during periods of low food availability and that the saved resources are invested in maintenance of the body until food resources are available for successful reproduction.
Protein restriction appears to have the same effects on rodents as calorie restriction, though it is less well-studied. An understanding of mechanisms for this marked effect on aging and cancer is becoming clearer and may in good part be due to reduced oxidative damage.
The suggestion that maintenance functions are enhanced in calorie-restricted rats thus resulting in less oxidative damage is supported by the findings of more efficient DNA repair, better coupled mitochondrial respiration and a delay in the age-dependent decline of antioxidant defenses.
The higher level of antioxidant defenses could also account for the enhanced immune response in restricted animals. We have recently shown that either calorie or protein restriction decreases the rate of accumulation of oxidized protein that accompanies aging in rats and preliminary results suggest a decrease in preneoplastic foci and oxidative lesions in DNA as well.
Thus, the overall effect of these enhanced maintenance activities appears to be a reduction in oxidative damage to DNA and protein, a decrease in DNA and protein lesions, and a decrease in somatic mutations. Markedly lower mitotic rates are observed in a variety of tissues in calorie restricted compared to ad libitum fed rodent, which may also contribute to the decrease in tumor incidence.
Click here for more about Glutathione and its benefits to our body.
*Dietary restriction activates the pituitary adrenocorticotropic axis resulting in a decrease in the release of reproductive and mitogenic hormones. Decreases in mitogenic hormones such as insulin, TSH, growth hormone, estrogen, and prolactin decrease the likelihood of hormone-induced cancers, as has been shown in various animal studies. This is consistent with suppression of mitogenic hormones and decreased proto-oncogene expression. The lowered incidence of mammary tumors observed in calorie-restricted rats has been attributed to reduced circulating levels of the mammotropic hormones estrogen and prolactin.
Click here for more about Glutathione and its benefits to our body.
Aging and Dietary Restriction
Evolutionary biologists have argued that aging is inevitable because of several tradeoffs. One tradeoff is that a considerable proportion of an animal's resources is devoted to reproduction at a cost to maintenance, which means that the maintenance of somatic tissues is less than that required for indefinite survival. Of the vast array of maintenance processes that are necessary to sustain normal function in somatic cells, those that defend the cell against metabolism derived oxidants are likely to play an important role.
Metabolism has costs: oxidants by-products of normal energy metabolism extensively damage DNA, proteins, and other molecules in the cell, and this damage accumulates with age.
Another tradeoff is that nature selects for many genes that have immediate survival value, but that may have long term deleterious consequences. The oxidative burst from phagocytic cells, for example, protects against death from bacterial and viral infections, but contributes to DNA damage, mutation, and cancer.
Click here for more about Glutathione and its benefits to our body.
Metabolism has costs: oxidants by-products of normal energy metabolism extensively damage DNA, proteins, and other molecules in the cell, and this damage accumulates with age.
Another tradeoff is that nature selects for many genes that have immediate survival value, but that may have long term deleterious consequences. The oxidative burst from phagocytic cells, for example, protects against death from bacterial and viral infections, but contributes to DNA damage, mutation, and cancer.
Click here for more about Glutathione and its benefits to our body.
Thursday, February 7, 2008
Oxidation and Damage to DNA, Protein, and Lipids
Oxidative damage to DNA, proteins and other macromolecules accumulates with age and has been postulated to be a major, but not the only, type of endogenous damage leading to aging. Superoxide(O2.-), hydrogen peroxide (H2O2), and hydroxyl radical (.OH), which are mutagens produced by radiation, are also by-products of normal metabolism. Lipid peroxidation gives rise to mutagenic lipid epoxides, lipid hydroperoxides, lipid alkoxyl and peroxyl radicals, and enals ([[alpha]], ß-unsaturated aldehydes). Singlet oxygen, a high energy and mutagenic form of oxygen, can be produced by transfer of energy from light, the respiratory burst from neutrophils, or lipid peroxidation.
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Animals have numerous antioxidants defenses, but since these defenses are not perfect, some DNA is oxidized. Oxidatively damaged DNA is repaired by enzymes that excise the lesions, which are then excreted in the urine. We have developed methods to assay several of these excised damaged bases in the urine of rodents and humans almost all of which appear as the free base from repair by glycosylases. We estimate that the number of oxidative hits to DNA per cell per day is about l00,000 in the rat and about l0,000 in the human. DNA repair enzymes efficiently remove most, but not all, of the lesions formed. Oxidative lesions in DNA accumulate with age, so that by the time a rat is old (2 years) it has about two million DNA lesions per cell, which is about twice that in a young rat. Mutations also accumulate with age . For example, the somatic mutation frequency in human lymphocytes, of which the contribution of oxidative DNA lesions is unknown, is about nine times greater in elderly people than in neonates. The importance of oxidative DNA lesions in cancer and aging is underscored by the existence of specific repair glycosylases that excise these lesions from DNA. In the case of 8-hydroxy-2'-deoxyguanosine, a lesion formed from oxidative damage to guanine residues in DNA, loss of a specific glycosylase activity leads to an appreciable increase in the spontaneous mutation rate, indicating the intrinsic mutagenic potential of this DNA lesion. Many other oxidative DNA lesions are likely to be important as well.
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Mitochondrial DNA (mtDNA) from rat liver has more than ten times the level of oxidative DNA damage than does nuclear DNA from the same tissue. This increase may be due to a lack of mtDNA repair enzymes, lack of histones protecting mtDNA, and the proximity of mtDNA to oxidants generated during oxidative phosphorylation. The cell defends itself against this high rate of damage by a constant turnover of mitochondria, thus presumably removing those damaged mitochondria that produce increased oxidants. Despite this turnover, oxidative lesions appear to accumulate with age in mtDNA at a higher rate than in nuclear DNA.
Oxidative damage could also account for the mutations in mtDNA that accumulate with age
Endogenous oxidants also damage proteins. Stadtman and his colleagues have shown that the proteolytic enzymes that hydrolyze oxidized proteins are not sufficient to prevent an age-associated accumulation of oxidized proteins. In two human diseases associated with premature aging, Werner's syndrome and progeria, oxidized proteins accumulate at a much higher rate than is normal. Fluorescent age pigments, which are thought to be due in part to cross-links between protein and lipid peroxidation products, also accumulate with aging.
Click here for more about Glutathione and its benefits to our body.
Animals have numerous antioxidants defenses, but since these defenses are not perfect, some DNA is oxidized. Oxidatively damaged DNA is repaired by enzymes that excise the lesions, which are then excreted in the urine. We have developed methods to assay several of these excised damaged bases in the urine of rodents and humans almost all of which appear as the free base from repair by glycosylases. We estimate that the number of oxidative hits to DNA per cell per day is about l00,000 in the rat and about l0,000 in the human. DNA repair enzymes efficiently remove most, but not all, of the lesions formed. Oxidative lesions in DNA accumulate with age, so that by the time a rat is old (2 years) it has about two million DNA lesions per cell, which is about twice that in a young rat. Mutations also accumulate with age . For example, the somatic mutation frequency in human lymphocytes, of which the contribution of oxidative DNA lesions is unknown, is about nine times greater in elderly people than in neonates. The importance of oxidative DNA lesions in cancer and aging is underscored by the existence of specific repair glycosylases that excise these lesions from DNA. In the case of 8-hydroxy-2'-deoxyguanosine, a lesion formed from oxidative damage to guanine residues in DNA, loss of a specific glycosylase activity leads to an appreciable increase in the spontaneous mutation rate, indicating the intrinsic mutagenic potential of this DNA lesion. Many other oxidative DNA lesions are likely to be important as well.
Click here for more about Glutathione and its benefits to our body.
Mitochondrial DNA (mtDNA) from rat liver has more than ten times the level of oxidative DNA damage than does nuclear DNA from the same tissue. This increase may be due to a lack of mtDNA repair enzymes, lack of histones protecting mtDNA, and the proximity of mtDNA to oxidants generated during oxidative phosphorylation. The cell defends itself against this high rate of damage by a constant turnover of mitochondria, thus presumably removing those damaged mitochondria that produce increased oxidants. Despite this turnover, oxidative lesions appear to accumulate with age in mtDNA at a higher rate than in nuclear DNA.
Oxidative damage could also account for the mutations in mtDNA that accumulate with age
Endogenous oxidants also damage proteins. Stadtman and his colleagues have shown that the proteolytic enzymes that hydrolyze oxidized proteins are not sufficient to prevent an age-associated accumulation of oxidized proteins. In two human diseases associated with premature aging, Werner's syndrome and progeria, oxidized proteins accumulate at a much higher rate than is normal. Fluorescent age pigments, which are thought to be due in part to cross-links between protein and lipid peroxidation products, also accumulate with aging.
Oxidants, Antioxidants, and the Degenerative Diseases of Aging
Metabolism, like other aspects of life, involves trade-offs. Oxidant by-products of normal metabolism cause extensive damage to DNA, protein, and lipid. We argue that this damage (the same as that produced by radiation) is a major contributor to aging and to degenerative diseases of aging such as cancer, cardiovascular disease, immune system decline, brain dysfunction and cataracts. Antioxidant defenses against this damage include ascorbate, tocopherol and carotenoids. Fruits and vegetables are the principal source of ascorbate and carotenoids and are one source of tocopherol. Low dietary intake of fruits and vegetables doubles the risk of most types of cancer as compared to high intake and also markedly increases the risk of heart disease and cataracts. Since only 9% of Americans eat the recommended five servings of fruits and vegetables per day the opportunity for improving health by improving diet is great.
The degenerative diseases associated with aging include cancer, cardiovascular disease, immune system decline, brain dysfunction, and cataracts. The functional degeneration of somatic cells during aging appears, in good part, to contribute to these diseases. The relationship between cancer and age in various mammalian species illustrates this point. Cancer increases with about the fifth power of age in both short-lived species, such as rats, and in long-lived species, such as humans.
Thus a marked decrease in age-specific cancer rates has accompanied the marked increase in life span that has occurred in 60 million years of mammalian evolution: i.e., cancer rates are high in a two year old rat, but low in a two year old human. One important factor in longevity appears to be basal metabolic rate, which is about seven times higher in a rat than a human and which could markedly affect the level of endogenous oxidants and other mutagens produced as by-products of metabolism. The level of oxidative DNA damage appears to be roughly related to metabolic rate in a number of mammalian species (cancer/mutation/endogenous DNA adducts/oxygen radicals)
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By Bruce N. Ames*, Mark K. Shigenaga, and Tory M. Hagen
The degenerative diseases associated with aging include cancer, cardiovascular disease, immune system decline, brain dysfunction, and cataracts. The functional degeneration of somatic cells during aging appears, in good part, to contribute to these diseases. The relationship between cancer and age in various mammalian species illustrates this point. Cancer increases with about the fifth power of age in both short-lived species, such as rats, and in long-lived species, such as humans.
Thus a marked decrease in age-specific cancer rates has accompanied the marked increase in life span that has occurred in 60 million years of mammalian evolution: i.e., cancer rates are high in a two year old rat, but low in a two year old human. One important factor in longevity appears to be basal metabolic rate, which is about seven times higher in a rat than a human and which could markedly affect the level of endogenous oxidants and other mutagens produced as by-products of metabolism. The level of oxidative DNA damage appears to be roughly related to metabolic rate in a number of mammalian species (cancer/mutation/endogenous DNA adducts/oxygen radicals)
Click here for more about Glutathione and its benefits to our body.
By Bruce N. Ames*, Mark K. Shigenaga, and Tory M. Hagen
Cysteine, Glutamic Acid and Glycine- immune system
Glutathione is a tri-peptide composed of three amino acids: Cysteine, Glutamic Acid and Glycine.
Glutathione and the enzymes it forms, such as GTH peroxidase, are essential to all life and are found in tissues of virtually all plants and animals. GTH is present in all human cells, with the highest levels found in the liver, the lenses of the eyes, pancreas, spleen and kidneys.
Glutathione acts as a powerful antioxidant, a key protector against all types of pollution and is effective in preventing aging. It protects DNA and RNA from free-radical damage.
Glutathione also protects against cellular peroxidation caused by exposure to pesticides, plastics, benzene and carbon tetrachloride, as well as heavy metals, cigarette smoke, smog, drugs, solvents, dyes, phenols and nitrates.
Glutathione works to inhibit the formation of free radicals, dangerous agents that suppress the immune system and promote the formation of mutagens and carcinogens.
Free radicals also speed up the aging process, and it is due to this powerful antioxidant activity that Glutathione is considered useful in the prevention and treatment of a wide range of degenerative diseases.
Studies at the Louisville School of Medicine have clearly shown that Glutathione possesses the unique ability to slow the aging process. While Glutathione aids in the protection of all cells and membranes, a study at Harvard Medical School found that Glutathione is especially able to enhance immune system cells, protecting against damage from radiation and helping to reduce the side effects of chemotherapy and x-rays and alcohol. As a detoxifier of metals and drugs, Glutathione also aids in the treatment of blood and liver disorders.
As individuals grow older, Glutathione levels drop, and the ability to detoxify free radicals decreases.
It can protect against cadmium, copper, and acetaminophen (the active agent in Tylenol) toxicity. Glutathione aids the liver in detoxification, slows the aging process, helps the cardiovascular and immune system, and is helpful in preventing or treating many other health conditions.
Supplementation may prevent, or be helpful with, the following conditions:
Aging
Alcoholism
Asthma
Atherosclerosis (heart disease)
Cancer
Cataracts
Dizziness
Hepatitis
Immunodepression (immune function)
Infertility (male)
Memory Loss (Alzheimer's disease, dementia)
Osteoarthritis
Parkinson's Disease
Peptic Ulcers
Click here for more about Glutathione and its benefits to our body.
Glutathione and the enzymes it forms, such as GTH peroxidase, are essential to all life and are found in tissues of virtually all plants and animals. GTH is present in all human cells, with the highest levels found in the liver, the lenses of the eyes, pancreas, spleen and kidneys.
Glutathione acts as a powerful antioxidant, a key protector against all types of pollution and is effective in preventing aging. It protects DNA and RNA from free-radical damage.
Glutathione also protects against cellular peroxidation caused by exposure to pesticides, plastics, benzene and carbon tetrachloride, as well as heavy metals, cigarette smoke, smog, drugs, solvents, dyes, phenols and nitrates.
Glutathione works to inhibit the formation of free radicals, dangerous agents that suppress the immune system and promote the formation of mutagens and carcinogens.
Free radicals also speed up the aging process, and it is due to this powerful antioxidant activity that Glutathione is considered useful in the prevention and treatment of a wide range of degenerative diseases.
Studies at the Louisville School of Medicine have clearly shown that Glutathione possesses the unique ability to slow the aging process. While Glutathione aids in the protection of all cells and membranes, a study at Harvard Medical School found that Glutathione is especially able to enhance immune system cells, protecting against damage from radiation and helping to reduce the side effects of chemotherapy and x-rays and alcohol. As a detoxifier of metals and drugs, Glutathione also aids in the treatment of blood and liver disorders.
As individuals grow older, Glutathione levels drop, and the ability to detoxify free radicals decreases.
It can protect against cadmium, copper, and acetaminophen (the active agent in Tylenol) toxicity. Glutathione aids the liver in detoxification, slows the aging process, helps the cardiovascular and immune system, and is helpful in preventing or treating many other health conditions.
Supplementation may prevent, or be helpful with, the following conditions:
Aging
Alcoholism
Asthma
Atherosclerosis (heart disease)
Cancer
Cataracts
Dizziness
Hepatitis
Immunodepression (immune function)
Infertility (male)
Memory Loss (Alzheimer's disease, dementia)
Osteoarthritis
Parkinson's Disease
Peptic Ulcers
Click here for more about Glutathione and its benefits to our body.
Wednesday, February 6, 2008
Inflammation and fever
Inflammation is a nonspecific response to infection or tissue injury. The four signs of the inflammatory response are redness, swelling, heat, and pain.
Inflammation begins when cells release certain cytokines, including interleukin (IL)-1, IL-6, and tumor necrosis factor-alpha (TNF-alpha ) (Janeway CA et al 1999; Beers MB 2004).
Inflammation is a process by which the body’s white blood cells and chemicals protect us from infection and foreign substances such as bacteria and viruses.
In some diseases, however, the body’s defense system (immune system) inappropriately triggers an Inflammation response when there are no foreign substances to fight off. In these diseases, called autoimmune diseases, the body’s normally protective immune system causes damage to its own tissues. The body responds as if normal tissues are infected or somehow abnormal.
Click here for more information about Glutathione and its benefits in our immune system.
Inflammation begins when cells release certain cytokines, including interleukin (IL)-1, IL-6, and tumor necrosis factor-alpha (TNF-alpha ) (Janeway CA et al 1999; Beers MB 2004).
Inflammation is a process by which the body’s white blood cells and chemicals protect us from infection and foreign substances such as bacteria and viruses.
In some diseases, however, the body’s defense system (immune system) inappropriately triggers an Inflammation response when there are no foreign substances to fight off. In these diseases, called autoimmune diseases, the body’s normally protective immune system causes damage to its own tissues. The body responds as if normal tissues are infected or somehow abnormal.
Click here for more information about Glutathione and its benefits in our immune system.
Physical and chemical barriers
The body's first lines of defense are the skin and mucous membranes, which prevent the entrance of many pathogens.
There are many secondary barriers.
For example, tears, sweat, and saliva combat some bacteria, and the hydrochloric acid and protein-digesting enzymes secreted by the stomach are lethal to many, but not all, pathogens (Janeway CA et al 1999; Beers MB 2004).
Click here for more information about Glutathione and its benefits in our immune system.
THE MAX GXL is a PATENTED High Performance Formula which:Dramatically Raises Your Energy LevelSlows Down The Aging ProcessStrengthens Your Immune SystemFights Inflammation and Diseases of AgingImproves Athletic Performance & RecoveryDetoxify Your Body
There are many secondary barriers.
For example, tears, sweat, and saliva combat some bacteria, and the hydrochloric acid and protein-digesting enzymes secreted by the stomach are lethal to many, but not all, pathogens (Janeway CA et al 1999; Beers MB 2004).
Click here for more information about Glutathione and its benefits in our immune system.
THE MAX GXL is a PATENTED High Performance Formula which:Dramatically Raises Your Energy LevelSlows Down The Aging ProcessStrengthens Your Immune SystemFights Inflammation and Diseases of AgingImproves Athletic Performance & RecoveryDetoxify Your Body
Sunday, February 3, 2008
Cause of Parkinson Disease
Although Parkinson’s disease can occur from viral infections or exposure to environmental toxins, such as pesticides (gardeners and farmers are more prone to Parkinson's disease).
The causes of the majority of cases are not well known. Scientists suspect that oxidative damage to neurons in the substantia nigra could well be one of the major causes, particularly due to the depletion of the antioxidants glutathione.
People who sustain substantial head injuries face an increased risk of developing Parkinson’s disease years later.
The cause of Parkinson's disease is unknown.
Many researchers believe that several factors combined are involved: free radicals, accelerated aging, environmental toxins, and genetic predisposition.
It may be that free radicals—unstable and potentially damaging molecules that lack on electron—are involved in the degeneration of dopamine-producing cells.
Free radicals add an electron by reacting with nearby molecules in a process called oxidation, which can damage nerve cells.
Chemicals called antioxidants normally protect cells from oxidative stress and damage. If antioxidative action fails to protect dopamine-producing nerve cells, they could be damaged and, subsequently, Parkinson’s disease could develop.
Dysfunctional antioxidative mechanisms are associated with older age as well, suggesting that the acceleration of age-related changes in dopamine production may be a factor.
Exposure to an environmental toxin, such as a pesticide, that inhibits dopamine production and produces free radicals and oxidation damage may be involved.
The causes of the majority of cases are not well known. Scientists suspect that oxidative damage to neurons in the substantia nigra could well be one of the major causes, particularly due to the depletion of the antioxidants glutathione.
People who sustain substantial head injuries face an increased risk of developing Parkinson’s disease years later.
The cause of Parkinson's disease is unknown.
Many researchers believe that several factors combined are involved: free radicals, accelerated aging, environmental toxins, and genetic predisposition.
It may be that free radicals—unstable and potentially damaging molecules that lack on electron—are involved in the degeneration of dopamine-producing cells.
Free radicals add an electron by reacting with nearby molecules in a process called oxidation, which can damage nerve cells.
Chemicals called antioxidants normally protect cells from oxidative stress and damage. If antioxidative action fails to protect dopamine-producing nerve cells, they could be damaged and, subsequently, Parkinson’s disease could develop.
Dysfunctional antioxidative mechanisms are associated with older age as well, suggesting that the acceleration of age-related changes in dopamine production may be a factor.
Exposure to an environmental toxin, such as a pesticide, that inhibits dopamine production and produces free radicals and oxidation damage may be involved.
Tuesday, January 22, 2008
Effect of glutathione depletion on antioxidant enzymes in the epididymis, seminal vesicles, and liver and on spermatozoa motility in the aging rat
Effect of glutathione depletion on antioxidant enzymes in the epididymis, seminal vesicles, and liver and on spermatozoa motility in the aging brown Norway rat.
by: EV Zubkova, B Robaire - Biol Reprod, Vol. 71, No. 3. (September 2004), pp. 1002-1008.
Reactive oxygen species (ROS) play a role in male infertility, where excessive amounts impair spermatozoal motility.
Epididymal antioxidant enzymes protect spermatozoa from oxidative damage in the epididymal lumen. Antioxidant secretions from the seminal vesicle protect spermatozoa after ejaculation.
As it is known that with age there is increased generation of ROS, the goals of this study were to determine how aging affects the response of antioxidant enzymes in the epididymis, seminal vesicles, and liver to l-buthionine-S,R-sulfoximine (BSO) mediated glutathione (GSH) depletion, and to examine the impact of GSH depletion on motility parameters of spermatozoa from the cauda epididymidis in young (4-mo-old) and old (21-mo-old) rats.
Levels of GSH and glutathione disulfide (GSSG), as well as activities of glutathione peroxidase, glutathione reductase, catalase, and superoxide dismutase, were measured in the caput, corpus and cauda epididymidis, seminal vesicles, and liver.
Spermatozoal motility was assessed by computer-assisted sperm analysis. Significant age-related changes in antioxidant enzyme activities were found in the liver and cauda epididymidis.
Glutathione age was most evident in the cauda epididymidis, seminal vesicles, and liver, where antioxidant enzyme activities changed significantly.
Additionally, spermatozoa motility was adversely affected after BSO treatment in both age groups, but significantly more so in older animals.
In summary, the male reproductive tissues and liver undergo age-related changes in antioxidant enzyme activities and in their response to GSH depletion.
THE MAX GXL is a PATENTED High Performance Formula which:Dramatically Raises Your Energy LevelSlows Down The Aging ProcessStrengthens Your Immune SystemFights Inflammation and Diseases of AgingImproves Athletic Performance & RecoveryDetoxify Your Body
by: EV Zubkova, B Robaire - Biol Reprod, Vol. 71, No. 3. (September 2004), pp. 1002-1008.
Reactive oxygen species (ROS) play a role in male infertility, where excessive amounts impair spermatozoal motility.
Epididymal antioxidant enzymes protect spermatozoa from oxidative damage in the epididymal lumen. Antioxidant secretions from the seminal vesicle protect spermatozoa after ejaculation.
As it is known that with age there is increased generation of ROS, the goals of this study were to determine how aging affects the response of antioxidant enzymes in the epididymis, seminal vesicles, and liver to l-buthionine-S,R-sulfoximine (BSO) mediated glutathione (GSH) depletion, and to examine the impact of GSH depletion on motility parameters of spermatozoa from the cauda epididymidis in young (4-mo-old) and old (21-mo-old) rats.
Levels of GSH and glutathione disulfide (GSSG), as well as activities of glutathione peroxidase, glutathione reductase, catalase, and superoxide dismutase, were measured in the caput, corpus and cauda epididymidis, seminal vesicles, and liver.
Spermatozoal motility was assessed by computer-assisted sperm analysis. Significant age-related changes in antioxidant enzyme activities were found in the liver and cauda epididymidis.
Glutathione age was most evident in the cauda epididymidis, seminal vesicles, and liver, where antioxidant enzyme activities changed significantly.
Additionally, spermatozoa motility was adversely affected after BSO treatment in both age groups, but significantly more so in older animals.
In summary, the male reproductive tissues and liver undergo age-related changes in antioxidant enzyme activities and in their response to GSH depletion.
THE MAX GXL is a PATENTED High Performance Formula which:Dramatically Raises Your Energy LevelSlows Down The Aging ProcessStrengthens Your Immune SystemFights Inflammation and Diseases of AgingImproves Athletic Performance & RecoveryDetoxify Your Body
Monday, January 21, 2008
Glutathione - MaxGXL
Supplements Containing Glutathione Alone Will NOT Increase The Body’s GSH Levels!It is pointless to purchase supplements that merely contain glutathione, because the digestive system breaks down ingested glutathione and it will not be absorbed into your system.
MaxGXL™ provides the proper nutrients needed to promote the body's own ability to manufacture and absorb glutathione. MaxGXL™ also aids in liver support by destroying environmental poisons helping the liver to function as the main production site and storehouse for glutathione.
THE MAX GXL is a PATENTED High Performance Formula which:
Dramatically Raises Your Energy Level
Slows Down The Aging Process
Strengthens Your Immune System
Fights Inflammation and Diseases of Aging
Improves Athletic Performance & Recovery
Detoxify Your Body
MaxGXL™ provides the proper nutrients needed to promote the body's own ability to manufacture and absorb glutathione. MaxGXL™ also aids in liver support by destroying environmental poisons helping the liver to function as the main production site and storehouse for glutathione.
THE MAX GXL is a PATENTED High Performance Formula which:
Dramatically Raises Your Energy Level
Slows Down The Aging Process
Strengthens Your Immune System
Fights Inflammation and Diseases of Aging
Improves Athletic Performance & Recovery
Detoxify Your Body
Glutathione Fact 2 - slow down the aging process
Glutathione has been shown to slow down the aging process, detoxify and improve liver function, strengthen the immune system, and reduce the chances of developing cancer.
Glutathione also works to help improve mental functions, increase energy, improve concentration, permit increased exercise, and improve heart and lung function - just to name a few.
Among the uses that have been reported for glutathione are:
treatment of poisoning, particularly heavy metal poisons
treatment of idiopathic pulmonary firbosis
increasing the effectiveness and reducing the toxicity of cis-platinum, a chemo drug used to treat breast cancer
treating Parkinson's disease
lowering blood pressure in patients with diabetes
increasing male sperm counts in humans and animals
treatment of liver cancer
treatment of sickle cell anemia
Glutathione also works to help improve mental functions, increase energy, improve concentration, permit increased exercise, and improve heart and lung function - just to name a few.
Among the uses that have been reported for glutathione are:
treatment of poisoning, particularly heavy metal poisons
treatment of idiopathic pulmonary firbosis
increasing the effectiveness and reducing the toxicity of cis-platinum, a chemo drug used to treat breast cancer
treating Parkinson's disease
lowering blood pressure in patients with diabetes
increasing male sperm counts in humans and animals
treatment of liver cancer
treatment of sickle cell anemia
Glutathione Fact 1 - produced naturally in our cells
Glutathione (GSH) is a small protein produced naturally in our cells when certain required elements are present
It functions both as an antioxidant and an antitoxin and is a major defense system against illness and aging.
Our glutathione level actually indicates our state of health and can predict longevity. Although there are more than 60,000 published papers on the beneficial effects of glutathione replacement, it is still largely ignored by mainstream medicine.
In the near future the importance of glutathione will be widely recognized because it has the ability to boost the immune system and fight off the damage of free radicals on the cells.
Glutathione's three major roles in the body are summarized by the letters A-B-C.
- Anti-oxidant- Blood Booster- Cell Detoxifier
It functions both as an antioxidant and an antitoxin and is a major defense system against illness and aging.
Our glutathione level actually indicates our state of health and can predict longevity. Although there are more than 60,000 published papers on the beneficial effects of glutathione replacement, it is still largely ignored by mainstream medicine.
In the near future the importance of glutathione will be widely recognized because it has the ability to boost the immune system and fight off the damage of free radicals on the cells.
Glutathione's three major roles in the body are summarized by the letters A-B-C.
- Anti-oxidant- Blood Booster- Cell Detoxifier
Friday, January 18, 2008
HOW TO GET GLUTATHIONE INTO YOUR CELLS
Glutathione is made up of three amino acids, glycine, glutamic acid and cysteine. Each cell produces its own GSH according to need within itself.
The determining factor as to how much it can make to ultimately keep levels up to the needed 70% in the active form...is determined by the availability of the amino acid cysteine. The production of cysteine becomes less and less efficient as we get older going into a steady decline by around age 55 onward.
This is just at a time when its most needed to protect our brains and neurological system from syndromes such as Alzheimer's Disease and dementia, which are now becoming more and more common.
Before we go further, let me explain "active" form (technically called the "reduced" form) and "inactive" (technically called the "oxidized" form) forms of glutathione. The active form is the one that can perform all the functions listed above act as a powerful antioxidant, work to combat diseases of aging and a potent player in detoxification. It is often shown in written works as "GSH".
The inactive form is present when GSH has done its work donating electrons and needs to regenerate itself which it can do. Its usually shown as GSSG (lacking the H..because it gave it away to stabilize some other molecule).
In a healthy situation, the percentage of GSH is about 90% with only 10% of Glutathione existing as GSSG. When these ratios change and there is more and more GSSG present, that is when the cell becomes sick, and is vulnerable to attack from toxins and microbes. If GSH falls below 70%...the cells/organ/person is in HUGE trouble!
There is one provison to this situation. If GSH is "used" to clear up a situation from our own metabolism, then it can regenerate itself just fine. HOWEVER if its used to combat "xenobiotics" or toxins from the outside of our bodies, such as chemicals, heavy metals, toxins, etc., then it cant regenerate.
This is where we run into trouble today as our food is not replenishing us the way it should and supplementing for Glutathione is tricky
Glutathione's three major roles in the body are summarized by the letters A-B-C.
- Anti-oxidant- Blood Booster- Cell Detoxifier
Learn more about Glutathione click here
The determining factor as to how much it can make to ultimately keep levels up to the needed 70% in the active form...is determined by the availability of the amino acid cysteine. The production of cysteine becomes less and less efficient as we get older going into a steady decline by around age 55 onward.
This is just at a time when its most needed to protect our brains and neurological system from syndromes such as Alzheimer's Disease and dementia, which are now becoming more and more common.
Before we go further, let me explain "active" form (technically called the "reduced" form) and "inactive" (technically called the "oxidized" form) forms of glutathione. The active form is the one that can perform all the functions listed above act as a powerful antioxidant, work to combat diseases of aging and a potent player in detoxification. It is often shown in written works as "GSH".
The inactive form is present when GSH has done its work donating electrons and needs to regenerate itself which it can do. Its usually shown as GSSG (lacking the H..because it gave it away to stabilize some other molecule).
In a healthy situation, the percentage of GSH is about 90% with only 10% of Glutathione existing as GSSG. When these ratios change and there is more and more GSSG present, that is when the cell becomes sick, and is vulnerable to attack from toxins and microbes. If GSH falls below 70%...the cells/organ/person is in HUGE trouble!
There is one provison to this situation. If GSH is "used" to clear up a situation from our own metabolism, then it can regenerate itself just fine. HOWEVER if its used to combat "xenobiotics" or toxins from the outside of our bodies, such as chemicals, heavy metals, toxins, etc., then it cant regenerate.
This is where we run into trouble today as our food is not replenishing us the way it should and supplementing for Glutathione is tricky
Glutathione's three major roles in the body are summarized by the letters A-B-C.
- Anti-oxidant- Blood Booster- Cell Detoxifier
Learn more about Glutathione click here
How much glutathione is enough?
Unfortunately, there is no Recommended Dietary Allowance (RDA) for glutathione to indicate how much is enough.
One hundred milligrams each day is the usual recommended dosage for glutathione.
THE MAX GXL is a PATENTED High Performance Formula which:
Dramatically Raises Your Energy Level
Slows Down The Aging Process
Strengthens Your Immune System
Fights Inflammation and Diseases of Aging
Improves Athletic Performance & Recovery
Detoxifies Your Body
One hundred milligrams each day is the usual recommended dosage for glutathione.
THE MAX GXL is a PATENTED High Performance Formula which:
Dramatically Raises Your Energy Level
Slows Down The Aging Process
Strengthens Your Immune System
Fights Inflammation and Diseases of Aging
Improves Athletic Performance & Recovery
Detoxifies Your Body
Glutathione peroxidase (GSH) is your body’s most abundant natural antioxidant.
GSH protects your vision, boosts your immune system, helps turn carbohydrates into energy, and prevents the buildup of oxidized fats that may contribute to atherosclerosis.
Glutathione is a compound classified as a tripeptide made of three amino acids: cysteine, glutamic acid, and glycine. Glutathione is also found in every part of the body, especially the lungs, intestinal tract, and liver.
The body produces and stores the largest amounts of GSH in the liver, where it is used to detoxify harmful compounds so that they can be removed from the body through the bile.
The liver also supplies GSH directly to red and white blood cells in the bloodstream; it helps keep red blood and white blood cells healthy to maximize the disease-fighting power of the immune system.
Glutathione also appears to have an anti-aging affect on the body. GSH levels decline with age, and a lack of Glutathione has been shown to leave the body more vulnerable to damage by free radicals, thus speeding up oxidation (wearing down) of the body.
A Glutathione deficiency can have a devastating effect on the nervous system, causing such symptoms as lack of balance and coordination, mental disorders, and tremors.
Any illness (even a bad cold), chronic disorders such as asthma and rheumatoid arthritis, injury, or heavy exposure to pollutants can cause a GSH deficiency.
This is because your body uses more GSH when it is supporting white blood cells and ridding the body of toxins.
Glutathione is found in almost all fruits and vegetables. Acorn squash, asparagus, avocado, cantaloupe, grapefruit, okra, orange, peach, potato, spinach, strawberries, tomato, watermelon, and zucchini are all good sources of GSH. Some vegetables, such as broccoli, cabbage, Brussels sprouts, cauliflower, kale, and parsley, not only provide GSH, but also actually stimulate the body produce more of this powerful antioxidant.
Cooking destroys a lot of the Glutathione in fresh fruits and vegetables, so you can get the most GSH from these foods by eating them raw or steamed.
Eating foods high in glutamine, such as lean meats, eggs, wheat germ, and whole grains, can also stimulate the liver to produce more GSH.
There is no Recommended Dietary Allowance (RDA) for GSH, but supplements have no known harmful side effects. Glutathione supplements can be expensive, but there is some question about the body’s ability to absorb GSH efficiently in supplemental form. If you want to take GSH supplements, just make sure to take them with meals to maximize absorption.
Click here for more about Glutathione and its benefits to our body.
Glutathione is a compound classified as a tripeptide made of three amino acids: cysteine, glutamic acid, and glycine. Glutathione is also found in every part of the body, especially the lungs, intestinal tract, and liver.
The body produces and stores the largest amounts of GSH in the liver, where it is used to detoxify harmful compounds so that they can be removed from the body through the bile.
The liver also supplies GSH directly to red and white blood cells in the bloodstream; it helps keep red blood and white blood cells healthy to maximize the disease-fighting power of the immune system.
Glutathione also appears to have an anti-aging affect on the body. GSH levels decline with age, and a lack of Glutathione has been shown to leave the body more vulnerable to damage by free radicals, thus speeding up oxidation (wearing down) of the body.
A Glutathione deficiency can have a devastating effect on the nervous system, causing such symptoms as lack of balance and coordination, mental disorders, and tremors.
Any illness (even a bad cold), chronic disorders such as asthma and rheumatoid arthritis, injury, or heavy exposure to pollutants can cause a GSH deficiency.
This is because your body uses more GSH when it is supporting white blood cells and ridding the body of toxins.
Glutathione is found in almost all fruits and vegetables. Acorn squash, asparagus, avocado, cantaloupe, grapefruit, okra, orange, peach, potato, spinach, strawberries, tomato, watermelon, and zucchini are all good sources of GSH. Some vegetables, such as broccoli, cabbage, Brussels sprouts, cauliflower, kale, and parsley, not only provide GSH, but also actually stimulate the body produce more of this powerful antioxidant.
Cooking destroys a lot of the Glutathione in fresh fruits and vegetables, so you can get the most GSH from these foods by eating them raw or steamed.
Eating foods high in glutamine, such as lean meats, eggs, wheat germ, and whole grains, can also stimulate the liver to produce more GSH.
There is no Recommended Dietary Allowance (RDA) for GSH, but supplements have no known harmful side effects. Glutathione supplements can be expensive, but there is some question about the body’s ability to absorb GSH efficiently in supplemental form. If you want to take GSH supplements, just make sure to take them with meals to maximize absorption.
Click here for more about Glutathione and its benefits to our body.
Thursday, January 17, 2008
The 3 Distinct Benefits of Naturally Produced Glutathione
Glutathione: The Master Antioxidant
Antioxidants participate directly in the destruction of reactive oxygen compounds called free radicals. These by-products of a cell’s normal function can’t be avoided, but exposure to ultraviolet radiation from the sun or other sources promotes their emergence.
Free radicals have been linked to muscle fatigue during exercise and aging.
For this reason, the body is equipped with a variety of antioxidants. Vitamins C and E are natural antioxidants but do not occur naturally in the body.
These and other antioxidants actually depend on natural glutathione to function properly.
This is why Glutathione is called “The Master Antioxidant”.
Glutathione : Food for the Immune system
Glutathione helps build your Immune system resistance and improve your chances of staying healthy.
Lymphocytes are cells of your Immune system. Glutathione is essential for lymphocytes to increase in number, produce antibodies, and function efficiently.
Glutathione: A Cellular Level Detoxifier
Our food and water sources are becoming increasingly contaminated with chemicals, as is the air that we breathe.
Supplemental Detoxifier such as Glutathione help to counter the effects of the toxins we inhale and ingest.
By physically binding to toxic compounds in cells, Glutathione helps make them soluble - and harmless. The body can then eliminate these disarmed toxins in the bile and urine.
Click here for more about Glutathione and its benefits to our body.
Antioxidants participate directly in the destruction of reactive oxygen compounds called free radicals. These by-products of a cell’s normal function can’t be avoided, but exposure to ultraviolet radiation from the sun or other sources promotes their emergence.
Free radicals have been linked to muscle fatigue during exercise and aging.
For this reason, the body is equipped with a variety of antioxidants. Vitamins C and E are natural antioxidants but do not occur naturally in the body.
These and other antioxidants actually depend on natural glutathione to function properly.
This is why Glutathione is called “The Master Antioxidant”.
Glutathione : Food for the Immune system
Glutathione helps build your Immune system resistance and improve your chances of staying healthy.
Lymphocytes are cells of your Immune system. Glutathione is essential for lymphocytes to increase in number, produce antibodies, and function efficiently.
Glutathione: A Cellular Level Detoxifier
Our food and water sources are becoming increasingly contaminated with chemicals, as is the air that we breathe.
Supplemental Detoxifier such as Glutathione help to counter the effects of the toxins we inhale and ingest.
By physically binding to toxic compounds in cells, Glutathione helps make them soluble - and harmless. The body can then eliminate these disarmed toxins in the bile and urine.
Click here for more about Glutathione and its benefits to our body.
Wednesday, January 16, 2008
Substances that Boost Glutathione Levels and Protect Brain Cells
Taking Glutathione itself as a supplement does not boost cellular Glutathione levels, since it breaks down in the digestive tract before it reaches the cells.
However, intravenous Glutathione therapy and Glutathione precursors or dietary supplements are effective in boosting intracellular levels ofGlutathione.
Intravenous Glutathione Injections:
Intravenous Glutathione injections have been shown to produce amazing and rapid results, in patients with Parkinson's disease. Followingeven a single dosage of intravenous Glutathione, many of the symptoms of Parkinson's disease rapidly improve, often in as little as 15 minutes.
Glutathione Precursors:
In the Alzheimer's study conducted by Welsh GP, Andrew McCaddon, adding theGlutathione precursor, N-acetyl-cysteine (NAC) to a protocol thatlowered homocysteine levels by simple supplementation with B12 and folate, resulted in prompt, striking, and sustained clinical improvement in nearly all the patients.
Cucurmin (turmeric):
Studies have shown that the Indian curry spice, cucurmin, has neuroprotective effects because of its ability to induce the enzyme, hemeoxygenase-1(HO-1), which protects neurons exposed to oxidant stress. Treatment of brain cells called astrocytes, with curcumin, increases expression of HO-1 protein as well as Glutathione S-transferase.
Proc Natl Acad Sci U S A. 2003 Jun 24;100(13):7919-24. Epub 2003 Jun 05.
Am J Geriatr Psychiatry. 2003 Mar-Apr;11(2):246-9
Can Curry Protect Against Alzheimer's?; American Physiological Society (APS) Press Release; 16-Apr-2004
Glutathione's three major roles in the body are summarized by the letters A-B-C.
- Anti-oxidant- Blood Booster- Cell Detoxifier
THE MAX GXL is a PATENTED High Performance Formula which:
Dramatically Raises Your Energy Level
Slows Down The Aging Process
Strengthens Your Immune System
Fights Inflammation and Diseases of Aging
Improves Athletic Performance & Recovery
Detoxifies Your Body
However, intravenous Glutathione therapy and Glutathione precursors or dietary supplements are effective in boosting intracellular levels ofGlutathione.
Intravenous Glutathione Injections:
Intravenous Glutathione injections have been shown to produce amazing and rapid results, in patients with Parkinson's disease. Followingeven a single dosage of intravenous Glutathione, many of the symptoms of Parkinson's disease rapidly improve, often in as little as 15 minutes.
Glutathione Precursors:
In the Alzheimer's study conducted by Welsh GP, Andrew McCaddon, adding theGlutathione precursor, N-acetyl-cysteine (NAC) to a protocol thatlowered homocysteine levels by simple supplementation with B12 and folate, resulted in prompt, striking, and sustained clinical improvement in nearly all the patients.
Cucurmin (turmeric):
Studies have shown that the Indian curry spice, cucurmin, has neuroprotective effects because of its ability to induce the enzyme, hemeoxygenase-1(HO-1), which protects neurons exposed to oxidant stress. Treatment of brain cells called astrocytes, with curcumin, increases expression of HO-1 protein as well as Glutathione S-transferase.
Proc Natl Acad Sci U S A. 2003 Jun 24;100(13):7919-24. Epub 2003 Jun 05.
Am J Geriatr Psychiatry. 2003 Mar-Apr;11(2):246-9
Can Curry Protect Against Alzheimer's?; American Physiological Society (APS) Press Release; 16-Apr-2004
Glutathione's three major roles in the body are summarized by the letters A-B-C.
- Anti-oxidant- Blood Booster- Cell Detoxifier
THE MAX GXL is a PATENTED High Performance Formula which:
Dramatically Raises Your Energy Level
Slows Down The Aging Process
Strengthens Your Immune System
Fights Inflammation and Diseases of Aging
Improves Athletic Performance & Recovery
Detoxifies Your Body
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